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In conclusion, a HIF-mediated shift toward improved energy supply may protect against acute tubular injury in various forms of AKI. The traditional discrimination between ischemic and toxic forms of AKI has been challenged because an increasing amount of evidence suggests that renal hypoxia is a common denominator in AKI of different etiologies.HIFs are heterodimers of a constitutive β subunit, HIF-β (ARNT), and one of three oxygen-dependent α-subunits, HIF-1α, HIF-2α, and HIF-3α.This to inform you that by clicking on ‘Accept’ you will be accessing a web application operated by a third party namely MDI India.Such content is provided only for the convenience of the Customers and Birla Sun Life Insurance Company Limited (BSLI) does not control such websites and these are approximate locations and there may be variations in the accuracy.This method is used for less temperature sensitive items such as lab ware and animal health products, bulky and/or heavy items The adenovirus E1B protein is a viral homolog of the Bcl-2 family of proteins that are involved in regulating cell death.A family of interacting proteins, which are designated Nip or Bnip and include Nip1, Nip2, Nip3 and Nix, associate with both the E1B protein and Bcl-2 proteins to mediate apoptotic signaling.
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Consequently, maneuvers of preconditional HIF activation are utilized to ameliorate AKI.
Indeed, many of these attempts are successful but the majority are conducted in ischemia-reperfusion injury.